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Dicarbonyls and advanced glycation end-products in the development of diabetic complications and targets for intervention

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The potential molecular mechanisms underlying these effects included disturbances in redox homeostasis (as evidence by the enhanced production of ROS and reactive nitrogen species), failure of enzymatic and nonenzymatic cytoprotective mechanisms involving the glutathione system as well as nuclear factor kappa B (NF-B) and fibroblast growth factor 2-mediated pathways, and impaired mitochondrial function

doi: 10.1155/2017/3720380 210 NurmasitohTSariDCRPartadiredjaG